Sunday, February 9, 2014

Activist Post - Vaccines and Shaken Baby Syndrome Webinar Feb. 5, 2014

Vaccines and Shaken Baby Syndrome Webinar Feb. 5, 2014

Catherine J. Frompovich
Activist Post

Vaccine apologists believe vaccines can cause no harm, something that is as far from the facts as any legitimate science ever should proclaim! The U.S. CDC’s VAERS reporting system documents vaccine damage claims, which are estimated to represent only one to ten (1:10) percent of the actual damages caused by vaccines.

One especially ‘visceral’ medico-legal form of damage is Shaken Baby Syndrome (SBS). Numerous parents have been legally prosecuted for shaking their babies to death whereas in reality, the damage done to the child’s brain was caused by toxic vaccine ingredients.

One has to realize and understand that for a true SBS event to have occurred, the child’s neck should be broken! For the amount of force and pressure to be exerted on the brain to cause the swelling and encephalopathies that result in SBS, there must be physical damage, especially neck trauma! Ironically, most cases of SBS do not present any physical damage, and yet parents are prosecuted with SBS.

Saturday, January 18, 2014

A Critical Review of the Classic Metaphyseal Lesion: Traumatic or Metabolic?

David M. Ayoub, Charles Hyman, Marta Cohen and Marvin Miller


OBJECTIVE. The purpose of this study was to review the hypothesis that classic metaphyseal lesions represent traumatic changes in abused infants and compare these lesions with healing rickets.

MATERIALS AND METHODS. Using a PubMed search, a multidisciplinary team reviewed studies that reported the histopathologic correlation of classic metaphyseal lesions. Selective studies of growth plate injury and rickets were cross-referenced.

RESULTS. Nine identified classic metaphyseal lesion studies were performed by the same principal investigator. Control subjects were inadequate. Details of abuse determination and metabolic bone disease exclusion were lacking. The presence of only a single radiology reviewer prevented establishment of interobserver variability. Microscopy was performed by two researchers who were not pathologists. Classic metaphyseal lesions have not been experimentally reproduced and are unrecognized in the accidental trauma literature. The proposed primary spongiosa location is inconsistent with the variable radiographic appearances. Classic metaphyseal lesions were not differentiated from tissue processing artifacts. Bleeding and callus were uncommon in spite of the vascular nature of the metaphysis. The conclusion that excessive hypertrophic chondrocytes secondary to vascular disruption were indicative of fracture healing contradicts the paucity of bleeding, callus, and periosteal reaction. Several similarities exist between classic metaphyseal lesions and healing rickets, including excessive hypertrophic chondrocytes. “Bucket-handle” and “corner fracture” classic metaphyseal lesions resemble healing rickets within the growth plate and the perichondrial ring, respectively. The age of presentation was more typical of bone fragility disorders, including rickets, than reported in prior child abuse series.

CONCLUSION. The hypothesis that classic metaphyseal lesions are secondary to child abuse is poorly supported. Their histologic and radiographic features are similar to healing infantile rickets. Until classic metaphyseal lesions are experimentally replicated and independently validated, their traumatic origin remains unsubstantiated.